The present study has examined the fine structure of freeze-fractured humanplatelets.
2
P-LPA added to humanplatelets caused aggregation without the need for ADP.
3
We recently demonstrated that smoking cessation causes temporary hyper-aggregability of humanplatelets.
4
The data provide evidence for the direct activation by thrombin of phospholipase D in humanplatelets.
5
We found that humanplatelets express 284 miRNAs.
Uso de human platelet en inglés
1
We studied the potential effects of NmU on humanplatelet activation.
2
The cells were successfully expanded in humanplatelet plasma supernatant, while maintaining their biological properties.
3
Previous reports on the direct effects of ethanol on humanplatelet aggregation function have been inconsistent.
4
Proper expression of the humanplatelet fibrinogen receptor is necessary for the maintenance of normal hemostasis.
5
These results indicate that adenosine and NECA increase humanplatelet metabolism, whereas PIA has an opposite effect.
6
Indole-3-carbinol significantly inhibited collagen-induced platelet aggregation in humanplatelet rich plasma (PRP) in a concentration-dependent manner.
7
In vitro humanplatelet activation assays revealed that rs1354034 is highly correlated with humanplatelet activation by ADP.
8
All the tested substances inhibit the humanplatelet aggregation induced by ADP and arachidonic acid in a broad concentration range.
9
The effects of divalent cations on humanplatelet vasopressin receptor binding characteristics and effects of receptor occupancy on endogenous protein phosphorylation were investigated.
10
A series of new 1,3,4-thiadiazine derivatives have been synthesized and their effect on the humanplatelet aggregation in vitro has been studied.
11
These results indicate that age and gender must be considered in designing clinical studies on 5HT and in evaluating humanplatelet 5HT kinetics.
12
The present study has examined the fine structure of freeze-fractured humanplatelets.
13
P-LPA added to humanplatelets caused aggregation without the need for ADP.
14
We recently demonstrated that smoking cessation causes temporary hyper-aggregability of humanplatelets.
15
The data provide evidence for the direct activation by thrombin of phospholipase D in humanplatelets.